Hepatocellular Carcinoma Cell – Timquinone selectively

Timquinone selectively induces hepatocellular cancer cell apoptosis in synergy with state clinical and p53 therapy. We also found that Hep3B cells with p53-zero status are more sensitive to QT than HepG2 and SMMC-7721 cells with wild type p53. Thus, p53 siRNA significantly increased jamming in HepG2 cells at QT-induced apoptosis measured by caspase 3 and PARP division. Timquinone selectively induces apoptosis in hepatocellular cancer cells in synergy with clinical therapy. To summarize, we found that TQ synergistically increases the anticancer activity of DOX and DDP, and that loss of p53 sensitizes GCC cells to apoptosis induced by TQ. TQ showed selective death for HCC HepG2 and SMMC-7721 cells, but relatively low toxicity for HL-7702 cells of normal liver. The information contained in this document does not diagnose, treat, facilitate or prevent any disease or illness. It is recommended that you seek the advice of a qualified medical professional before beginning any natural, integrative, or traditional treatment. It is important to note that when used with DOX or DDP, TQ showed synergistic inhibition of HCC cells, but not HL-7702 cells. We studied the cytotoxic effect of TQ, alone or in combination with DDP and DOX, on HCC cells. Over 500 pages with alternatives and information on naturopathy

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